Aliases:
ECCMO
ECMO

Topic aliases are alternate phrasings for a particular topic.


In patients with acidemia and impaired myocardial function VV ECMO may improve the patient's metabolic derangement resulting in improved myocardial function. This may avoid the need for VA ECMO.
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Qualifying criteria :
A-a gradient >600 for >4hrs;
oxygenation index >40 in 3 of 5 post-ductal gas 30min apart;
acute deterioration (PaO2 <40mmHg >2hrs,
pH <7.25 >2hrs,
intractable hypotension. #ref

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Causes of hypoxia on VV ECMO.

1. recirculation
2. Cardiac output > ECMO flow
3. Oxygenator failure

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Differential hypoxia may occur in patients on peripheral VA ECMO with poor lungs. Pulse oximetry on the right arm will better reflect cerebral oxygenation.
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Example of clot

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Will require measurement of ACT. Ensure functioning equipment available.

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Extension tubing under drapes to foot of bed to anaesthetist/intensivist.

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Calcium replacement may be prudent as the patient is connected to the ECMO circuit as the prime blood is citrated and may precipitate hypocalcemia and particularly in neonates, reduced myocardial contractility.

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Heparin 100u/kg prior to first cannulation. Ask surgeon when to administer.

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A means of assessing whether peripheral VA ECMO is providing adequately oxygenated blood to the brain is to look at the oximetry in the right hand.

Adequately oxygenated blood in the right hand suggests that oxygenated blood from the arterial ECMO cannula is reaching the right subclavian artery and hence likely the brain via the carotids.

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If considering defibrillation whilst on ECMO consider the risk that the physical jolt may disrupt cannula connections.

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Ensure blood is crossmatched & available in the room. For paediatrics, have blood prepared in a 50ml syringe to administer as needed.

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Normal ranges of plasma free Hb in patients on ECMO

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Precalculate allowable blood-loss (ABL) and transfuse appropriately

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Fever is an unreliable sign of infection in patients on ECMO as temperature is regulated by the circuit.

#TD

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